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2nd International Conference and Expo on Lipids: Metabolism, Nutrition & Health, will be organized around the theme “Solving the Impetus of Innovations in Lipid World”

Lipids 2016 is comprised of 14 tracks and 88 sessions designed to offer comprehensive sessions that address current issues in Lipids 2016.

Submit your abstract to any of the mentioned tracks. All related abstracts are accepted.

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The Injectable Drug Delivery Market by Formulation (Liposomes,  Nanoparticles and Microspheres), Devices and Therapeutics [Diabetes and Oncology] - Global Forecasts to 2017, dissects and contemplates the real market drivers, restrictions, and chances in North America, Europe, Asia-Pacific, and Rest of the World. Worldwide injectable medication conveyance advancements market over the gauge time of 2012 to 2017. The worldwide injectable medication conveyance advancements business sector was esteemed at $22.5 billion in 2012; it is relied upon to reach $43.3 billion by 2017 at a CAGR of 14.0% from 2012 to 2017. Injectable medication conveyance advancements are the mix of two noteworthy portions; gadgets and definitions. This business sector understanding report on Liposomes gives knowledge into worldwide Liposomes market. The concentrate additionally gives data with respect to items and innovations in the field of Liposomes. Business profiles of 46 noteworthy organizations are examined in the report. The report covers more than 330 organizations that are occupied with Liposomes research and supply of items and/or administrations. The worldwide liposomes' business sector is anticipated at a CAGR of 15%, through 2005-2015. Clinical drugs that connect with membrane lipids and that modify the composition and structure of cell membranes can transform the localization and/or activity of membrane proteins. In general, adjustments in membrane lipid structure are emulated in changes in membrane lipid activity. Several drugs used to fight against cancer, cardiovascular diseases and obesity and other pathologies, determine lipid structure in plasma membrane and they produce a concomitant alteration in the localization and activity of signaling proteins. The recent studies have resulted in identifying specific disease-provoking gene mutations and have led to improved clinical and laboratory treatments, prenatal diagnosis in lipid related diseases.

Relevant Conferences

2nd International Conference on  Molecular Medicine and Diagnostics during September 26-28, 2016, Orlando, Florida, USAInternational Conference on Clinical Chemistry and Laboratory Medicine during October 17-18, 2016 Chicago, Illinois, USA; International Conference on Biochemistry during October 10-12, 2016 Kuala Lumpur, Malaysia; 2nd International Conference on Transcriptomics September 12-14, 2016 Philadelphia, Pennsylvania, USA; International Conference on Next Generation Sequencing November 17-18, 2016, Atlanta, Georgia, USA 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; 12th GERLI Lipidomics Meeting, "Microbe and Host Lipids" October 23-26, 2016, Toulouse, France

  • Track 1-1Membrane-lipid therapy
  • Track 1-2Genetic and Molecular basis of lipid regulation underlie lipid-related diseases
  • Track 1-3Lipid-antigens in Immunity
  • Track 1-4Lipid Metabolism, Inflamation in Chronic diseases
  • Track 1-5Lipids and oxidative stress
  • Track 1-6Gene regulation of lipid metabolism
  • Track 1-7Solid lipid nanoparticles and challenges

The total cost of reducing low-density lipoprotein includes the costs of physician services, lifestyle counseling, screening, case finding and monitoring, dietary and exercise modifications, medications, and treating side effects. The annual cost of statin drugs to reduce low-density lipoprotein cholesterol levels can range from $1,082 to $1,543 per year. The cost of follow-up or treatment-related appointments varies by type of provider, location, and practice setting. Although the cost of reducing low-density lipoprotein cholesterol levels can be high, it is much lower than the direct and indirect costs of cardiovascular disease research. Hyperlipoproteinemia is the lipid unsettling influence of significant pertinence clinically as a result of its link with an enlarged danger of atherosclerotic cardiovascular sickness. Hoisted plasma triglycerides and low-thickness lipoproteins are specifically connected with the danger of atherosclerotic coronary illness, in spite of the fact that not as free hazard variables. Interestingly, abnormal states of high-thickness lipoprotein cholesterol have been observed to be a defensive component for the advancement of that infection, so that diminished levels constitute a danger element. Atherosclerosis is described by vascular impediment from the stores of plaque, bringing about diminished blood stream. Plaque crack and the subsequent thrombosis might prompt sudden blockage of the corridors and cause heart assault. Plasma lipoproteins have generally been gathered into five noteworthy classes, in light of their light density: chylomicrons, low-thickness lipoproteins (VLDL), halfway thickness lipoproteins (IDL), low-thickness lipoproteins (LDL), and high-thickness lipoproteins (HDL). Since lipids are not solvent in blood, they are transported as lipoproteins after response with water-dissolvable proteins in the blood. Lipids in the blood are consumed by liver cells to give vitality to cell capacities. Overabundance lipids in the blood are in the long run changed over into fat tissue. Unusually elevated amounts of triglycerides and cholesterol are speculated to be involved in solidifying of the corridors. In nondiabetic persons of ordinary weight, organization of insulin prompts an expansion in glucose up-take by insulin-delicate tissues, hindrance of lipolysis, and a reduction in serum levels of free unsaturated fats. Be that as it may, in insulin-safe states joined by hyperinsulinemia, for example, happen in heftiness and type II diabetes, there is imperviousness to brings about expanded fat breakdown and expanded serum levels of free unsaturated fats and glycerol. The expanded glycerol coming about because of lipolysis tends to drive gluconeogenesis by mass impact, along these lines prompting expanded glucose generation by the liver, further adding to hyperglycemia. Lipid stockpiling ailments are a gathering of acquired metabolic disorder in which hurtful measures of greasy materials (lipids) amass in different tissues and cells in the body. Lipid stockpiling ailments are acquired from one or both folks who convey an imperfect quality.

Relevant Conferences

2nd International Conference on Lipid Science & Technology October 03-05, 2016 Orlando, USA; InternationalConference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan;International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 6th International Conference on Metabolomics November 28-30, 2016 Atlanta, USA; Lipid andLipoprotein Metabolism in Chronic Disease Pathogenesis and Treatment, June 12-17, 2016 Waterville Valley, NH; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; 12th GERLI Lipidomics Meeting, "Microbe and Host Lipids" October 23-26, 2016, Toulouse, France

  • Track 2-1Lipoprotein Metabolism: Cellular and Molecular Mechanisms
  • Track 2-2Atherosclerosis-Lipoprotein-Related Mechanisms
  • Track 2-3LDL,HDL,VLDL
  • Track 2-4Lipid Metabolism in Diabetes
  • Track 2-5Lipids and Metabolic Diseases
  • Track 2-6Dyslipidemia and Myocardial ischemia
  • Track 2-7Blood lipid Metabolism – understandings and objectives
  • Track 2-8Environmental Influences on Lipid and Lipoprotein Metabolism
  • Track 2-9Nutrition, Physical Activity, Lipids

An individual lipid particle will stay in the annular shell around a protein for just a brief timeframe. Tying to the annular shell indicates generally minimal auxiliary specificity. And the annular lipid, there is proof for other lipid atoms bound between the transmembrane alpha-helices of the protein; these lipids are alluded to as non-annular lipids. Voltage-gated channels are key transducers of film potential changes into intracellular homeless people that start numerous physiological occasions. The conformational change mutilates the state of the channel proteins adequately such that the depression, or channel, opens to concede particle flood or efflux to happen over the layer, down its electrochemical angle. Lipid microarrays will give an incorporated learning base to the human lipidome. Exogenous fat is transported in chylomicrons from the digestive system to the liver. After passage in the circulatory system the chylomicrons are hydrolyzed by the endothelial-enslaved lipoprotein lipase. The chylomicron leftovers are quickly taken up into the liver by means of the LDL receptor and the LDL receptor-linked protein. Apolipoprotein E and lipoprotein lipase are the acknowledgment signals for these receptors. The liver uses the exogenous fat and can discharge surplus lipids by means of VLDL into the blood. The remaining VLDL leftovers can either be taken up into the liver or are hydrolyzed to LDL. Both these types of Hypercholesterolemia are the most successive and speak to significant danger elements for arteriosclerosis.

Relevant Conferences

6th Global Healthcare  & Fitness Summit, December 05-07, 2016 Philadelphia, Pennsylvania, USA; GlobalSummit  on Healthcare, November 17-19, 2016 Dubai UAE; 5th Asian Obesity Specialists and Endocrinologists Annual Meeting July 11-13, 2016 Kuala Lumpur, Malaysia; 5th International Conference on Agriculture & Horticulture June 27-29, 2016 Cape Town, South Africa; LIPID MAPS Annual Meeting 2016: Lipidomics Impact on Metabolic, Cancer, Cardiovascular and Inflammatory Diseases , May 17 - 18, 2016, La Jolla, CA, USA; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 3rd International 4 Corners of Cardiology Meeting, February 05-06 2016, Australia; 22nd International Symposium on Plant Lipids03-08 July 2016, Goettingen, Germany; 8th European Symposium on Plant Lipids 2-5 July 2017, Malmo, Sweden

  • Track 3-1Structure, energetics, and dynamics of lipid–protein interactions
  • Track 3-2Cell membrane receptors and Voltage gated channels
  • Track 3-3Micelles
  • Track 3-4Lipid-protein interactions in monolayers
  • Track 3-5Impact of membrane properties on interactions at the protein–lipid interface
  • Track 3-6Advances in Lipid-Protein Interactions

Today 66% of grown-ups and about 33% of youngsters battle with overweight and obesity. If heftiness rates stay reliable, by 2030, 51 percent of the populace will be obese. Twenty years prior, no state had a stoutness rate above 15 percent. Today there are 41 states with stoutness rates more than 25 percent, as indicated by the Trust for American's Health. Following 1980, the rate of weight in kids and young people has practically tripled. 72% of more established men and 67% of more seasoned ladies are currently overweight or stout. Heftiness is connected to more than 60 perpetual illnesses. By American Cancer Society, 572,000 Americans kick the bucket of tumor every year, around 33% of these growth passings are connected to overabundance body weight, poor sustenance and/or physical idleness. More than 75 percent of hypertension cases are specifically connected to stoutness. Roughly 66% of U.S. grown-ups with sort 2 diabetes are overweight or have heftiness.

Relevant Conferences

International Conference on Clinical Chemistry and Laboratory Medicine during October 17-18, 2016 Chicago, Illinois, USA; International Conference on Biochemistry during October 10-12, 2016 Kuala Lumpur, Malaysia; 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; World Congress on Human Genetics  November 07-08, 2016 Barcelona, Spain; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; Leucine Rich Repeat Kinase 2: Ten Years Along the Road to Therapeutic Intervention, Henley Business School, UK, 11-13 July 2016; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; International Conference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan

  • Track 4-1Obesity and Weight Management
  • Track 4-2Childhood Obesity and its Lifelong Effects
  • Track 4-3Health Risks Associated with Obesity
  • Track 4-4Obesity and Type 2 diabetes
  • Track 4-5Endocrine Disorders and Obesity
  • Track 4-6Obesity & Pregnancy
  • Track 4-7Diet and physical activity
  • Track 4-8Nutrition and Obesity Prevention
  • Track 4-9Current Research and Clinical Interventions

Understanding the mechanisms of intracellular trafficking and its interaction with other signaling molecules may lead to novel approaches in the treatment of a number of hematologic and other diseases. NIH has invested $127,980 in this specific area in 2015. Lipid rafts/caveolae flagon formed structures are rich in proteins and additionally lipids, for example, cholesterol and sphingolipids and have a few capacities in signal transduction. They assume a part in disease cells advancement, endocytosis and the uptake of pathogenic microorganisms and certain infections. Ponders that have illustrated the part of lipid pontoons in flagging by means of bioreceptor tyrosine kinases and G protein-coupled receptors. The inositol phospholipids frame the basic premise for a mind boggling interchange of flagging reactions created, most regularly, by receptor actuation and bringing about changes in Ca +2 , protein kinase falls, and particle channel/exchanger movement. Phosphatidylinositol (PI) itself is a negligible phospholipid constituent of all eukaryote plasma films. Despite the fact that the plasma layer might contain microdomains with an assortment of various lipid creations, cholesterol and sphingolipid rich microdomains, named lipid flatboats, have been the most strongly examined. Flatboats are hypothesized to direct protein–protein interactions by horizontally isolating proteins as per their partiality for requested layer areas. New endeavours need to create and test elective theories for lipid-intervened natural capacity are basic to propelling our comprehension of plasma layer areas and their parts in cell capacity. Cholesterol is an amphipathic particle like phospholipid moiety. It contains a hydrophobic interactions and hydrophilic interactions. Regardless of its little mass, cholesterol assumes a critical part in keeping up the trustworthiness of the film and is likewise included with cell to cell flagging procedure.

Relevant Conferences

2nd International Conference on Transcriptomics September 12-14, 2016 Philadelphia, Pennsylvania, USA; International Conference on Next Generation Sequencing November 17-18, 2016, Atlanta, Georgia, USA; 8th Global Cardiologists Annual Meeting, July 18-20 2016, Germany; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; Recent Advances in Lipoprotein Physiologyand Related Disease, Waterville Valley, NH, June 11-12, 2016; Contemporary Methodologies for Analysis andCharacterization of Lipids 23-24 August 2016, Stockholm, Sweden; International Symposium on Lipid Oxidationand Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; Third World Congress of Clinical Lipidology 10-12 February 2017, Brisbane, Australia; 13th Yeast Lipid Conference, 17-19 May 2017, Paris, France

  • Track 5-1Rafts/caveolae- roles in signal transduction
  • Track 5-2Phosphoinositide signalling and intracellular vesicle trafficking
  • Track 5-3Microdomains in the membrane landscape
  • Track 5-4Cholesterol and membrane integrity
  • Track 5-5Lipid phosphate phosphatases in cellular signaling
  • Track 5-6Cellular uptake and intracellular trafficking of long chain fatty acids
  • Track 5-7Bio active lipid signaling
  • Track 5-8Signaling by fatty-acylated and prenylated proteins
  • Track 5-9Non-vesicular trafficking
  • Track 5-10Trafficking of G protein–Coupled receptors

Today 66% of grown-ups and about 33% of youngsters battle with overweight and obesity. If heftiness rates stay reliable, by 2030, 51 percent of the populace will be obese. Twenty years prior, no state had a stoutness rate above 15 percent. Today there are 41 states with stoutness rates more than 25 percent, as indicated by the Trust for American's Health. Following 1980, the rate of weight in kids and young people has practically tripled. 72% of more established men and 67% of more seasoned ladies are currently overweight or stout. Heftiness is connected to more than 60 perpetual illnesses. By American Cancer Society, 572,000 Americans kick the bucket of tumor every year, around 33% of these growth passings are connected to overabundance body weight, poor sustenance and/or physical idleness. More than 75 percent of hypertension cases are specifically connected to stoutness. Roughly 66% of U.S. grown-ups with sort 2 diabetes are overweight or have heftiness.

Relevant Conferences

International Conference on Clinical Chemistry and Laboratory Medicine during October 17-18, 2016 Chicago, Illinois, USA; International Conference on Biochemistry during October 10-12, 2016 Kuala Lumpur, Malaysia; 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; World Congress on Human Genetics  November 07-08, 2016 Barcelona, Spain; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; Leucine Rich Repeat Kinase 2: Ten Years Along the Road to Therapeutic Intervention, Henley Business School, UK, 11-13 July 2016; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; International Conference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan

  • Track 6-1Renewable bioenergy using microorganisms
  • Track 6-2Bioenergy-oriented microbial lipid technology
  • Track 6-3Algae biofuels
  • Track 6-4Solid waste management
  • Track 6-5Production of Bioenergy and biochemicals from POME
  • Track 6-6BIofuels - Environment

The economic burden of lipid disorders is substantial because of the impact of lipid levels on the risk of cardiovascular disease and coronary heart disease events. The direct and indirect costs of all types of cardiovascular disease in 2015 were estimated to be $706.2 billion. The cost of cardiovascular disease exceeds that of any other high-cost medical conditions. For example, in 2008, the estimated total cost of all cancers was $228 billion and in 2007, the cost attributable to diabetes mellitus was $174 billion. The nature of fat is for the most part indicated by the relative substance of SFA, monounsaturated (MUFA), and polyunsaturated unsaturated fats (PUFA) including the extent or measure of vital unsaturated fats, that is, linoleic corrosive (LA) and α-linolenic corrosive (ALA),as well as the extent or measure of long-chain n-3 unsaturated fats (n-3 LCPUFA), that is, eicosapentaenoic corrosive (EPA) and docosahexaenoic corrosive (DHA). Coronary illness is perceived to be the reason for death for 80% of individuals with diabetes. Diabetes is treatable, however notwithstanding when glucose levels are under control it incredibly expands the danger of coronary illness and stroke. Hypertension has for quite some time been perceived as a noteworthy danger component for cardiovascular illness. Weight is a noteworthy danger element for cardiovascular disease and has been unequivocally connected with insulin resistance. Physical idleness is another modifiable real hazard component for insulin resistance and cardiovascular infection. Fish, plant, and nut oils are the essential dietary origin of omega-3 unsaturated fats. Eicosapentaenoic corrosive (EPA) and docosahexaenoic corrosive (DHA) are found in icy water fish, for example, salmon, mackerel, halibut, sardines, fish, and herring. Dietary admission of omega-3 unsaturated fats has declined by 80% amid the most recent 100 years, while admission of omega-6 unsaturated fats has extraordinarily expanded. Omega-3 unsaturated fats are cardioprotective for the most part because of helpful impacts on arrhythmiasatherosclerosis, irritation, and thrombosis

Relevant Conferences

5th International Conference on Agriculture & Horticulture June 27-29, 2016 Cape Town, South Africa; 2ndInternational Congress on Biofuels & Bioenergy, September 1-3, 2016, Sao Paulo, Brazil; Euro Global Summit on Biomass August 08-10, 2016 Birmingham, UK; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; Recent Advances in Lipoprotein Physiology and Related Disease, Waterville Valley, NH, June 11-12, 2016; Innovative Approaches towards a Sustainable Future 18-21 September 2016 Ghent, Belgium; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; 12thGERLI Lipidomics Meeting, "Microbe and Host Lipids" October 23-26, 2016, Toulouse, France; InternationalConference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan

  • Track 7-1Cardiometabolic Effects of Popular Diets
  • Track 7-2Diabetes and Heart Failure: The Joint Epidemics
  • Track 7-3Omega-3 Fatty Acids, Cardiovascular Health and Controversy
  • Track 7-4Novel Approaches to Treating dyslipidemia and Atherosclerosis
  • Track 7-5Medical and Surgical Approaches to Improving Heart Failure Outcomes
  • Track 7-6Latest Nutrition Trials in combating Cardio Vascular Disease
  • Track 7-7Stem Cell Repair in Models of Cardio Vascular Disease

Lipids play diverse roles in the normal functioning of the body. They serve as the structural building material of all cell membranes and organelles. They provide energy for living organisms - providing more than twice the energy content compared with carbohydrates and proteins on a weight basis. They function as molecular messengers and signalling molecules in the body.

Lipids are also biomarkers of disease and are involved in several pathological conditions. Lipids are also known to play a role in genetic modification and influence risk of chronic disease.

Some of the fatty acids need to be taken in diet. This includes essential fatty acids (EFAs), linoleic acid (LA, an omega-6 fatty acid, 18:2n-6), and a-linolenic acid (LNA, an omega-3 fatty acid, 18:3n-3). These help in formation of polyunsaturated fatty acids (PUFAs) used in cellular structures and as precursors for the biosynthesis of many of the body’s regulatory molecules like long-chain PUFAs, arachidonic acid,  eicosapentaenoic acid (EPA, 20:5n-3), and docosahexaenoic acid (DHA, 22:6n-3) and eicosanoids. DHA again is necessary for normal neural and retinal development in the infant and young child.

Relevant Conferences

International Conference on Clinical Chemistry and Laboratory Medicine during October 17-18, 2016 Chicago, Illinois, USA; International Conference on Biochemistry during October 10-12, 2016 Kuala Lumpur, Malaysia; 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; World Congress on Human Genetics  November 07-08, 2016 Barcelona, Spain; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; Leucine Rich Repeat Kinase 2: Ten Years Along the Road to Therapeutic Intervention, Henley Business School, UK, 11-13 July 2016; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; International Conference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan

  • Track 8-1Nutrition science
  • Track 8-2Macronutrients
  • Track 8-3Micronutrients
  • Track 8-4Metabolism and Nutrition Needs 

The fact that lipids are soluble in organic solvents, but insoluble in water, provides the food analyst with a convenient method of separating the lipid components in foods from water soluble components, such as proteins, carbohydrates and minerals. In fact, solvent extraction techniques are one of the most commonly used methods of isolating lipids from foods and of determining the total lipid content of foods.

The aim of all extraction procedures is to separate cellular or fluid lipids from the other constituents, proteins, polysaccharides, small molecules (amino acids, sugars...) but also to preserve these lipids for further analyses.

There is a great diversity of methodologies because biological tissues are not similar when considering their structure, texture, sensitivities and lipid contents. The ideal solvent for lipid extraction would completely extract all the lipid components from a sample, while leaving all the other components behind. In practice, the efficiency of solvent extraction depends on the polarity of the lipids present compared to that of the solvent.

Polar lipids (such as glycolipids or phospholipids) are more soluble in polar solvents (such as alcohols), than in non-polar solvents (such as hexane). On the other hand, non-polar lipids (such as triacylglycerols) are more soluble in non-polar solvents than in polar ones. The fact that different lipids have different polarities means that it is impossible to select a single organic solvent to extract them all. Thus the total lipid content determined by solvent extraction depends on the nature of the organic solvent used to carry out the extraction: the total lipid content determined using one solvent may be different from that determined using another solvent.

The high sensitivity of the analytical methods needed for low amounts of extracted lipids requires the use of very pure solvents and clean glassware. The chemical nature of the extracted lipids must also be taken into consideration. 

Relevant Conferences

2nd International Conference on  Molecular Medicine and Diagnostics during September 26-28, 2016, Orlando, Florida, USA; International Conference on Clinical Chemistry and Laboratory Medicine during October 17-18, 2016 Chicago, Illinois, USA; International Conference on Biochemistry during October 10-12, 2016 Kuala Lumpur, Malaysia; 2nd International Conference on Transcriptomics September 12-14, 2016 Philadelphia, Pennsylvania, USA; International Conference on Next Generation Sequencing November 17-18, 2016, Atlanta, Georgia, USA 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; 12th GERLI Lipidomics Meeting, "Microbe and Host Lipids" October 23-26, 2016, Toulouse, France

  • Track 9-1Mass spectrometry
  • Track 9-2MALDI-TOF
  • Track 9-3High Performance Liquid Chromatography
  • Track 9-4Imaging techniques
  • Track 9-5Informatics
  • Track 9-6Lipidomic profiling

Lipids are a diverse and ubiquitous group of compounds which have many key biological functions, such as acting as structural components of cell membranes, serving as energy storage sources and participating in cell signaling pathways. Lipids may be broadly defined small molecules that originate entirely or in part from two distinct as hydrophobic or amphipathic types of biochemical subunits or "building blocks": ketoacyl and isoprene groups. The  huge structural diversity found in lipids arises from the biosynthesis of various combinations of these building blocks. For example, glycerophospholipids are composed of a glycerol backbone linked to one of approximately 10 possible headgroups and also to 2 fatty acyl/alkyl chains, which in turn may have 30 or more different molecular structures. In practice, not all possible permutations are detected experimentally, due to chain preferences depending on the cell type and also to detection limits - nevertheless several hundred distinct glycerophospholipid molecular species have been detected in mammalian cells.

For years, reductionism derived from the "fluid mosaic" model of the cell membrane has portrayed membrane lipids as rather passive molecules that, whereas separating biologically relevant aqueous phases, provided an environment so that membrane proteins could fulfil the specificity and selectivity required for proper cell signaling. Whereas these roles for membrane lipids still stand, the structural diversity of lipids and their complex arrangement in supramolecular assemblies have expanded such limited, although fundamental roles. Growing developments in the field of membrane lipids help to understand biological phenomena at the nanoscale domain, and reveal this heterogeneous group of organic compounds as a long underestimated group of key regulatory molecules. 

Relevant Conferences

6th Global Healthcare  & Fitness Summit, December 05-07, 2016 Philadelphia, Pennsylvania, USA; GlobalSummit  on Healthcare, November 17-19, 2016 Dubai UAE; 5th Asian Obesity Specialists and Endocrinologists Annual Meeting July 11-13, 2016 Kuala Lumpur, Malaysia; 5th International Conference on Agriculture & Horticulture June 27-29, 2016 Cape Town, South Africa; LIPID MAPS Annual Meeting 2016: Lipidomics Impact on Metabolic, Cancer, Cardiovascular and Inflammatory Diseases , May 17 - 18, 2016, La Jolla, CA, USA; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 3rd International 4 Corners of Cardiology Meeting, February 05-06 2016, Australia; 22nd International Symposium on Plant Lipids03-08 July 2016, Goettingen, Germany; 8th European Symposium on Plant Lipids 2-5 July 2017, Malmo, Sweden

  • Track 10-1Fatty Acids
  • Track 10-2Triacylglycerol
  • Track 10-3Membrane Lipids
  • Track 10-4Lipid Vesicles

The role of oxidized lipids in cardiovascular diseases (CVD) has been investigated over the last three decades extensively. A number of studies have been carried out on the mechanisms, and pathways leading to the arterial atherosclerosis.

Lipid abnormalities play a critical role in the development of atherosclerosis.[3] Early experiments in animals demonstrated accelerated atherosclerosis with a high-cholesterol diet. This was followed by epidemiologic studies conducted in countries around the world that showed an increasing incidence of atherosclerosis when serum cholesterol concentrations were elevated.

Atherosclerosis is the leading cause of death in North America and within the next two decades will be the leading cause worldwide. Atherosclerosis is characterized by vascular obstruction from the deposits of plaque, resulting in reduced blood flow. Plaque rupture and the consequent thrombosis may lead to sudden blockage of the arteries and cause heart attack. High serum lipid levels, especially the elevated level of low-density lipoproteins (LDL), have been shown to be strongly related to the development of atherosclerosis. It is generally accepted that atherosclerotic lesions are initiated via an enhancement of LDL uptake by monocytes and macrophages. In the liver, uptake of plasma LDL is mediated via specific LDL receptors, but a scavenger receptor system is employed by macrophages. Plasma LDL must be modified prior to uptake by macrophages.

Relevant Conferences

2nd International Conference on Lipid Science & Technology October 03-05, 2016 Orlando, USA; InternationalConference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan;International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 6th International Conference on Metabolomics November 28-30, 2016 Atlanta, USA; Lipid andLipoprotein Metabolism in Chronic Disease Pathogenesis and Treatment, June 12-17, 2016 Waterville Valley, NH; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; 12th GERLI Lipidomics Meeting, "Microbe and Host Lipids" October 23-26, 2016, Toulouse, France

  • Track 11-1Triglyceride-rich lipoproteins
  • Track 11-2High-density lipoproteins (HDL)
  • Track 11-3Lp(a) lipoprotein
  • Track 11-4Low-density lipoprotein (LDL)

Essential oils have been used for more than 5,000 years. Their origin can be traced back to the ancient civilization of Mesopotamia. In later years the technology of obtaining these oils spread to Egypt, India, Greece and Rome. The most commonly used essential oils are lavender, chamomile, peppermint, tea tree oil, eucalyptus, geranium, jasmine, rose, lemon, orange, rosemary, frankincense, and sandalwood. Most of the trade in essential oils occurs in the European Union (EU), the North American Free Trade Area (NAFTA), South America and East Asia with very little or insignificant trade happening in Africa and in particular the SADC region. Over the past two decades there has been an increase in the amount of overall trade (imports and exports) in essential oils, from about just over US$616-million in 1990 to more than US$3.6-billion in 2005. The EU has inherently been the largest trader of essential oils. In 2005 it imported US$536m and exported US$765m of US$1.7bn and US $1.9 bn world imports and exports, respectively. In the same period the north Atlantic free Trade Area (nAfTA) imported US$378m worth of imports and exported US$476m. SADC’s trade, which the main area of focus in this report is insignificant in the context of world trade – just about 1% overall. In 2005 exports and imports amounted to US$15.4m and US $25.8m, respectively. The opportunities for the region to increase its share in world trade are plenty. This is despite the fact that big established multinational corporations (i.e. end-users) have developed solid commercial relationship with suppliers that have a proven record of supplying high quality products. Lipid Peroxidation by an unmistakable bit of UV light prompts an adjustment of the plant lipids. This change renders them solid inducers of heme oxygenase expression and blend of glutathione, the most important cellular cell reinforcement. Initiated lipid separates from plants prompt inborn cancer prevention agent and detoxificationpathways of skin cells. Most solid individuals can eat ALA from plant sources like chia and flaxseeds, and it will be changed into EPA and DHA. Just seven to 15 percent of the dietary ALA might be changed over to EPA. Considerably less is changed over to DHA. Furthermore, this is in a sound youngster. Soybean, canola, wheat germ and walnut oil all have a nice measure of omega-3's, yet much larger amounts of omega-6 unsaturated fats - and this proportion of omega-3's to omega-6's is imperative. Wellbeing specialists suggest an omega-6/omega-3 proportion of around 4:1. 

  • Track 12-1Anti-ageing
  • Track 12-2Plant Derived Omega-3 Fatty Acids- a healthy perspective
  • Track 12-3Plant lipids - Medical and Food Applications
  • Track 12-4Essential oils and aromatic flavours
  • Track 12-5Microbial Lipids
  • Track 12-6Function-spacer-lipid construct strategy
  • Track 12-7Lipids mediators in microbial pathogenesis
  • Track 12-8Anti MIcrobial Activity of Essential Oils

The compartmentation of impartial lipids in plants is for the most part connected with seed tissues, where triacylglycerols (TAGs) put away inside lipid beads (LDs) serve as a key physiological vitality and carbon hold amid post germinative development. Be that as it may, some non-seed tissues, for example, leaves, blooms and natural products, additionally blend and store TAGs, yet moderately little is thought about the development or capacity of LDs in these tissues. Portrayal of LD-related proteins, for example, oleosins, caleosins, and sterol dehydrogenases (steroleosins), has uncovered astonishing components of LD capacity in plants, including cell stress reactions, hormone flagging pathways, and different parts of plant development and improvement. In spite of the fact that oleosin and caleosin proteins are particular to plants, LD-related sterol dehydrogenases likewise are available in well evolved creatures, and in both plants and warm blooded animals these compounds have been appeared to be imperative in (steroid) hormone digestion system and flagging. Also, a few different proteins known not essential in LD biogenesis in yeasts and vertebrates are preserved in plants, recommending that in any event a few parts of LD biogenesis and/or capacity are developmentally monitored.

In most plants storage lipids are in the form of triglycerides. There are a very few examples of alternative forms of storage lipid in higher plants. The most known of these is the desert shrub, jojoba, which stores its seed lipid as a liquid wax. Storage lipids may be accumulated in one or both of the main types of seed tissue, embryo or endosperm. In oilseeds such as sunflower, linseed or rapeseed, the cotyledons of the embryo are the major sites of lipid accumulation. In species such as castor bean, coriander or carrot, the endosperm is the main site of lipid accumulation.

World Congress on Human Genetics  November 07-08, 2016 Barcelona, Spain; World Congress on Amino Acids and Proteins December 08-09, 2016 Dallas, Texas, USA; 2nd International Conference on Transcriptomics September 12-14, 2016 Philadelphia, Pennsylvania, USA; 5th Leipzig Symposium on, "Analytics and Technology" Leipzig, Germany, 9-10 March 2016; Organelle Crosstalk in Membrane Dynamicsand Cell Signalling, Edinburgh, UK; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Leucine Rich Repeat Kinase 2: Ten Years Along the Road to Therapeutic Intervention, Henley Business School, UK, 11-13 July 2016; 57th International Conference on the Bioscience of Lipids, Chamonix, France, September 23 - 27, 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA; International Conference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference on Metabolomics May 16-18, 2016 Osaka, Japan

  • Track 13-1Plant stress responses
  • Track 13-2Surface lipids: cutin, waxes and suberin
  • Track 13-3Sphingolipids, sterols, isoprenoids
  • Track 13-4Lipid Peroxidation

Lipidomics has been incredibly encouraged by late advances in, and novel utilizations of, electrospray ionization mass spectrometry (ESI/MS. Lipidomic concentrates on assume a fundamental part in characterizing thebiochemical metabolism of lipid-related sickness forms through distinguishing adjustments in cell lipid digestion system, trafficking and homeostasis. The two noteworthy stages at present utilized for lipidomic investigations are HPLC-MS and shotgun lipidomics. Late extension in exploration in the field of lipidomics has been driven by the advancement of new mass spectrometry and lipidomics devices and conventions for the distinguishing proof and measurement of sub-atomic lipids in complex networks. The utilization of electrospray ionization to unrefined lipid removes without earlier fractionation-the purported shotgun methodology is one such illustration, as it has maybe been more effectively connected in lipidomics than in some other Recent innovative advances in MS and chromatography have significantly upgraded the improvements and uses of metabolic profiling of various lipids in complex natural specimens. Lipidomics not just give experiences into the particular elements of lipid species in wellbeing and infection, yet will likewise distinguish potential biomarkers for building up preventive or restorative projects for human illness.

Relevant Conferences

Euro Global Summit and Expo on Biomass August 08-10, 2016 Birmingham, UK; 2nd International Conference on Lipid Science & Technology October 03-05, 2016 Orlando, USA; International Conference on Glycomics December 01-02, 2016 Atlanta, USA; 5th International Conference and Exhibition on Metabolomics May 16-18, 2016 Osaka, Japan; 6th International Conference and Exhibition on Metabolomics November 28-30, 2016 Atlanta, USA; Lipid and Lipoprotein Metabolism in Chronic Disease Pathogenesis and Treatment, June 12-17, 2016 Waterville Valley, NH; 4th International Conference on Integrative Biology July 18-20, 2016 Berlin, Germany; International Symposium on Lipid Oxidation and Antioxidants 5-7 June 2016, Porto, Portugal; Phosphatases and Signalling in Health and Disease, University of Bath, UK, 20-22 June 2016; 107th AOCS Annual Meeting & Expo, May 1 - 4, 2016 Salt Lake City, Utah, USA

  • Track 14-1Shotgun lipidomics
  • Track 14-2Lipidomics and Imaging Mass Spectrometry
  • Track 14-3Lipidomics and applications in disease biomarker discovery
  • Track 14-4Neurodegenerative diseases and lipidomic strategy